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author | Karl Berry <karl@freefriends.org> | 2011-05-12 22:55:47 +0000 |
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committer | Karl Berry <karl@freefriends.org> | 2011-05-12 22:55:47 +0000 |
commit | aaabc08a0813867da817735d0c417ef0e7c98d81 (patch) | |
tree | 0f57e3277c6cbc17945782bc48d1ce09f8973ee4 /Master/texmf-dist/doc/latex/unamthesis/testBib.bib | |
parent | a89412c0be73dfd0f50269a87d5cd92f541165cd (diff) |
new latex package unamthesis (12may11)
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diff --git a/Master/texmf-dist/doc/latex/unamthesis/testBib.bib b/Master/texmf-dist/doc/latex/unamthesis/testBib.bib new file mode 100644 index 00000000000..4b65ee25357 --- /dev/null +++ b/Master/texmf-dist/doc/latex/unamthesis/testBib.bib @@ -0,0 +1,351 @@ +% This file was created with JabRef 2.6. +% Encoding: Cp1252 + +@BOOK{Barabasi2002, + title = {Linked: The New Science of Networks}, + publisher = {Perseus Books Group}, + year = {2002}, + author = {Albert-L\'{a}szl\'{o} Barab\'{a}si}, + pages = {256}, + edition = {First}, + month = may, + isbn = {0738206679}, + owner = {jfreyre}, + timestamp = {2008.07.09} +} + +@ARTICLE{Freyre-Gonzalez2008, + author = {Julio Augusto Freyre-Gonz\'{a}lez and Jos\'{e} Antonio Alonso-Pav\'{o}n + and Luis Gerardo Treviño-Quintanilla and Julio Collado-Vides}, + title = {Functional architecture of \textit{Escherichia coli}: new insights + provided by a natural decomposition approach.}, + journal = {Genome Biol}, + year = {2008}, + volume = {9}, + pages = {R154}, + number = {10}, + month = {Oct}, + abstract = {ABSTRACT: BACKGROUND: Previous studies have used different methods + in an effort to extract the modular organization of transcriptional + regulatory networks. However, these approaches are not natural, as + they try to cluster strongly connected genes into a module or locate + known pleiotropic transcription factors in lower hierarchical layers. + Here, we unravel the transcriptional regulatory network of Escherichia + coli by separating it into its key elements, thus revealing its natural + organization. We also present a mathematical criterion, based on + the topological features of the transcriptional regulatory network, + to classify the network elements into one of two possible classes: + hierarchical or modular genes. RESULTS: We found that modular genes + are clustered into physiologically correlated groups validated by + a statistical analysis of the enrichment of the functional classes. + Hierarchical genes encode transcription factors responsible for coordinating + module responses based on general interest signals. Hierarchical + elements correlate highly with the previously studied global regulators, + suggesting that this could be the first mathematical method to identify + global regulators. We identified a new element in transcriptional + regulatory networks never described before: intermodular genes. These + are structural genes which integrate, at the promoter level, signals + coming from different modules, and therefore from different physiological + responses. Using the concept of pleiotropy, we have reconstructed + the hierarchy of the network and discuss the role of feedforward + motifs in shaping the hierarchical backbone of the transcriptional + regulatory network. CONCLUSIONS: This study sheds new light on the + design principles underpinning the organization of transcriptional + regulatory networks, showing a novel nonpyramidal architecture comprised + of independent modules globally governed by hierarchical transcription + factors, whose responses are integrated by intermodular genes.}, + doi = {10.1186/gb-2008-9-10-r154}, + owner = {jfreyre}, + pii = {gb-2008-9-10-r154}, + pmid = {18954463}, + timestamp = {2008.10.29}, + url = {http://dx.doi.org/10.1186/gb-2008-9-10-r154} +} + +@MISC{Freyre-Gonzalez2005, + author = {Julio A. Freyre-Gonz\'{a}lez and Jos\'{e} A. Alonso-Pav\'{o}n and + Daniel V\'{a}zquez-Hernandez and Mario Sandoval-Calderon and Mariana + Matus-Garc\'{\i}a and Ortega-del Vecchyo, Diego and Julio Collado-Vides}, + title = {Modular and hierarchical organization of the transcriptional regulatory + network of \textit{Escherichia coli} K-12}, + howpublished = {5th International Workshop on Bioinformatics and Systems Biology, + Poster Session, Berlín, Alemania}, + month = {August}, + year = {2005}, + abstract = {There are strong arguments that support the idea of modular organization + in the cell [3]. A module is defined as a group of correlated elements + that cooperate in a specific cellular function [3,1]. In genetic + networks, these modules are integrated by transcription factors (TFs) + and genes that act coordinately when specific stimuli are present. + In biological networks there exist global TFs that interact with + several elements of many modules. This makes difficult or impossible + to classify those TFs into a single module. Consequently, we may + classify the network’s elements into two groups: elements that belong + to modules (genes and local TFs, which will hereafter be called modular + elements), and elements that coordinate such modules in a hierarchical + fashion (global TFs and sigma factors, which will hereafter be called + control elements). This suggests that a methodology that will allow + for the classification of the network’s genes in one of the aforementioned + groups is required. + + Recently, topological analyses have suggested the existence of hierarchical + modularity in the transcriptional regulatory network (TRN) of E. + coli [2,6,5]. Nevertheless, these studies have neglected the importance + of classifying genes in modular and control elements, as well as + the existence of feedback circuits among them. Such feedback circuits + could be interpreted as a mechanism by which control elements retrieve + information about the status of genes in modules and, based on this + feedback signal, generate decisions about the fate of the cell. + + Assuming these hypotheses, in our laboratory, we are working on an + algorithm to propose a hierarchical structure of the TRN: + + 1. Using data from RegulonDB [7,4] we will decompose the network, + through the analysis of the node degree and clustering coefficient + distribution, into the aforementioned groups and temporally remove + the control elements. + + 2. Using Monica Riley’s gene functional assignations [8] we will analyze + the modules to determine whether they are physiologically correlated + or not. + + 3. Finally, we will add the removed control elements to infer the + hierarchical structure of the TRN. + + On this poster we will show the results obtained from applying this + methodology to the TRN of E. coli.}, + owner = {jfreyre}, + timestamp = {2008.11.09} +} + +@ARTICLE{Gottesman1984, + author = {S. Gottesman}, + title = {Bacterial regulation: global regulatory networks}, + journal = {Annu Rev Genet}, + year = {1984}, + volume = {18}, + pages = {415--441}, + doi = {10.1146/annurev.ge.18.120184.002215}, + keywords = {Aerobiosis; Anaerobiosis; Bacterial Proteins; Base Sequence; Cyclic + AMP; DNA Repair; Energy Metabolism; \textit{Escherichia coli}; Gene + Expression Regulation; Glucose; Heat-Shock Proteins; Nitrogen; Operon; + Phosphate; Receptors, Cyclic AMP; Repressor Proteins; s}, + owner = {jfreyre}, + pmid = {6099091}, + timestamp = {2008.02.05}, + url = {http://dx.doi.org/10.1146/annurev.ge.18.120184.002215} +} + +@ARTICLE{Griffith2002, + author = {Kevin L Griffith and Ishita M Shah and Todd E Myers and Michael C + O'Neill and Richard E Wolf}, + title = {Evidence for ``pre-recruitment'' as a new mechanism of transcription + activation in \textit{Escherichia coli}: the large excess of SoxS + binding sites per cell relative to the number of SoxS molecules per + cell}, + journal = {Biochem Biophys Res Commun}, + year = {2002}, + volume = {291}, + pages = {979--986}, + number = {4}, + month = {Mar}, + abstract = {In response to the oxidative stress imposed by redox-cycling compounds + like paraquat, \textit{Escherichia coli} induces the synthesis of + SoxS, which then activates the transcription of approximately 100 + genes. The DNA binding site for SoxS-dependent transcription activation, + the "soxbox," is highly degenerate, suggesting that the genome contains + a large number of SoxS binding sites. To estimate the number of soxboxes + in the cell, we searched the \textit{E. coli} genome for SoxS binding + sites using as query sequence the previously determined optimal SoxS + binding sequence. We found approximately 12,500 sequences that match + the optimal binding sequence under the conditions of our search; + this agrees with our previous estimate, based on information theory, + that a random sequence the size of the \textit{E. coli} genome contains + approximately 13,000 soxboxes. Thus, fast-growing cells with 4-6 + genomes per cell have approximately 65,000 soxboxes. This large number + of potential SoxS binding sites per cell raises the interesting question + of how SoxS distinguishes between the functional soxboxes located + within the promoters of target genes and the plethora of equivalent + but nonfunctional binding sites scattered throughout the chromosome. + To address this question, we treated cells with paraquat and used + Western blot analysis to determine the kinetics of SoxS accumulation + per cell; we also determined the kinetics of SoxS-activated gene + expression. The abundance of SoxS reached a maximum of 2,500 molecules + per cell 20 min after induction and gradually declined to approximately + 500 molecules per cell over the next 1.5 h. Given that activation + of target gene expression began almost immediately and given the + large disparity between the number of SoxS molecules per cell, 2,500, + and the number of SoxS binding sites per cell, 65,000, we infer that + SoxS is not likely to activate transcription by the usual "recruitment" + pathway, as this mechanism would require a number of SoxS molecules + similar to the number of soxboxes. Instead, we propose that SoxS + first interacts in solution with RNA polymerase and then the binary + complex scans the chromosome for promoters that contain a soxbox + properly positioned and oriented for transcription activation. We + name this new pathway "pre-recruitment."}, + doi = {10.1006/bbrc.2002.6559}, + institution = {Department of Biological Sciences, University of Maryland Baltimore + County, Baltimore, Maryland 21250, USA.}, + keywords = {Bacterial Proteins; Binding Sites; Blotting, Western; Cell Division; + DNA-Binding Proteins; \textit{Escherichia coli}; \textit{Escherichia + coli} Proteins; Gene Expression Regulation, Bacterial; Genome, Bacterial; + Kinetics; Numerical Analysis, Computer-Assisted; Oxidative Stress; + Paraquat; Protein Transport; Trans-Activation (Genetics); Trans-Activators; + Transcription Factors}, + owner = {jfreyre}, + pii = {S0006291X02965599}, + pmid = {11866462}, + timestamp = {2008.02.05}, + url = {http://dx.doi.org/10.1006/bbrc.2002.6559} +} + +@ARTICLE{Hartwell1999, + author = {L. H. Hartwell and J. J. Hopfield and S. Leibler and A. W. Murray}, + title = {From molecular to modular cell biology}, + journal = {Nature}, + year = {1999}, + volume = {402}, + pages = {C47--C52}, + number = {6761 Suppl}, + month = {Dec}, + abstract = {Cellular functions, such as signal transmission, are carried out by + 'modules' made up of many species of interacting molecules. Understanding + how modules work has depended on combining phenomenological analysis + with molecular studies. General principles that govern the structure + and behaviour of modules may be discovered with help from synthetic + sciences such as engineering and computer science, from stronger + interactions between experiment and theory in cell biology, and from + an appreciation of evolutionary constraints.}, + doi = {10.1038/35011540}, + institution = {Fred Hutchinson Cancer Center, Seattle, Washington 98109, USA.}, + keywords = {Action Potentials; Evolution; Forecasting; Models, Biological; Molecular + Biology}, + owner = {jfreyre}, + pmid = {10591225}, + timestamp = {2008.02.05}, + url = {http://dx.doi.org/10.1038/35011540} +} + +@ARTICLE{Keseler2005, + author = {Ingrid M Keseler and Julio Collado-Vides and Socorro Gama-Castro + and John Ingraham and Suzanne Paley and Ian T Paulsen and Martín + Peralta-Gil and Peter D Karp}, + title = {EcoCyc: a comprehensive database resource for \textit{Escherichia + coli}}, + journal = {Nucleic Acids Res}, + year = {2005}, + volume = {33}, + pages = {D334--D337}, + number = {Database issue}, + month = {Jan}, + abstract = {The EcoCyc database (http://EcoCyc.org/) is a comprehensive source + of information on the biology of the prototypical model organism + \textit{Escherichia coli} K12. The mission for EcoCyc is to contain + both computable descriptions of, and detailed comments describing, + all genes, proteins, pathways and molecular interactions in E.coli. + Through ongoing manual curation, extensive information such as summary + comments, regulatory information, literature citations and evidence + types has been extracted from 8862 publications and added to Version + 8.5 of the EcoCyc database. The EcoCyc database can be accessed through + a World Wide Web interface, while the downloadable Pathway Tools + software and data files enable computational exploration of the data + and provide enhanced querying capabilities that web interfaces cannot + support. For example, EcoCyc contains carefully curated information + that can be used as training sets for bioinformatics prediction of + entities such as promoters, operons, genetic networks, transcription + factor binding sites, metabolic pathways, functionally related genes, + protein complexes and protein-ligand interactions.}, + doi = {10.1093/nar/gki108}, + institution = {SRI International, 333 Ravenswood Avenue, Menlo Park, CA 94025, USA.}, + keywords = {Computational Biology; Databases, Genetic; \textit{Escherichia coli} + K12; \textit{Escherichia coli} Proteins; Gene Expression Regulation, + Bacterial; Genome, Bacterial; Genomics; Software; User-Computer Interface}, + owner = {jfreyre}, + pii = {33/suppl_1/D334}, + pmid = {15608210}, + timestamp = {2008.02.05}, + url = {http://dx.doi.org/10.1093/nar/gki108} +} + +@ARTICLE{Leskovec2008, + author = {Jure Leskovec and Eric Horvitz}, + title = {Planetary-scale views on an instant-messaging network}, + year = {2008}, + month = mar, + abstract = {We present a study of anonymized data capturing a month of high-level + communication activities within the whole of the Microsoft Messenger + instant-messaging system. We examine characteristics and patterns + that emerge from the collective dynamics of large numbers of people, + rather than the actions and characteristics of individuals. The dataset + contains summary properties of 30 billion conversations among 240 + million people. From the data, we construct a communication graph + with 180 million nodes and 1.3 billion undirected edges, creating + the largest social network constructed and analyzed to date. We report + on multiple aspects of the dataset and synthesized graph. We find + that the graph is well-connected and robust to node removal. We investigate + on a planetary-scale the oft-cited report that people are separated + by ``six degrees of separation'' and find that the average path length + among Messenger users is 6.6. We also find that people tend to communicate + more with each other when they have similar age, language, and location, + and that cross-gender conversations are both more frequent and of + longer duration than conversations with the same gender.}, + eprint = {arXiv:0803.0939v1 [physics.soc-ph]}, + keywords = {Physics - Physics and Society}, + owner = {jfreyre}, + timestamp = {2008.07.01}, + url = {http://arxiv.org/abs/0803.0939} +} + +@BOOK{Lipschutz1986, + title = {Estructura de Datos}, + publisher = {Mcgraw-Hill}, + year = {1986}, + author = {Seymour Lipschutz}, + pages = {352}, + series = {Serie Schaum}, + month = dec, + isbn = {0070380015}, + owner = {jfreyre}, + timestamp = {2008.07.09} +} + +@INCOLLECTION{Marconi1967, + author = {Guglielmo Marconi}, + title = {Wireless Telegraphic Communication}, + booktitle = {Physics 1901--1921}, + publisher = {Elsevier Publishing Company}, + year = {1967}, + series = {Nobel Lectures}, + pages = {196--222}, + address = {Amsterdam}, + owner = {jfreyre}, + timestamp = {2008.06.30} +} + +@INCOLLECTION{Neidhardt1996, + author = {Neidhardt, F. C. and Savageau, M.}, + title = {Regulation beyond the operon}, + booktitle = {Escherichia coli and Salmonella: Cellular and Molecular Biology}, + publisher = {American Society for Microbiology}, + year = {1996}, + editor = {Neidhardt, F. C.}, + pages = {1310--1324}, + address = {Washington D.C.}, + edition = {Second}, + owner = {jfreyre}, + timestamp = {2008.04.18} +} + +@BOOK{Nelson2000, + title = {Lehninger Principles of Biochemistry}, + publisher = {W. H. Freeman}, + year = {2000}, + author = {David L. Nelson and Michael M. Cox}, + pages = {1200}, + edition = {Third}, + month = feb, + isbn = {1572599316}, + owner = {jfreyre}, + timestamp = {2008.07.25} +} + |